Unity in strength storyo12/29/2023 ![]() The most alarming report to suggest lingering heart injury appeared in July in JAMA Cardiology. Many of them also had indicators of scarring and decreased right ventricle function. Fourteen patients had edema-fluid retention, which is the key sign of active inflammation on CMR. The patients had had symptoms including chest pain and palpitations for a median of 47 days by the time they underwent cardiac magnetic resonance (CMR) imaging. This unknown has made it hard to interpret some findings from recovered patients.Ī study published in May, for example, examined 26 patients discharged from a Wuhan hospital after recovering from moderate to severe COVID-19. In March, physicians in Italy determined that inflammation of the heart muscle and sac, known as myopericarditis, was likely behind extreme fatigue in an otherwise healthy 53-year-old woman with a positive SARS-CoV-2 test who had mild respiratory symptoms and fever a week earlier.Įxperts haven’t reached consensus on how long viral myocarditis takes to resolve, in part because sophisticated imaging tools and protocols for accurately diagnosing it are relatively new in addition, the duration of clinical symptoms may not match serological or imaging biomarkers. The effects of cardiac inflammation in COVID-19 are wide-ranging and, for some, appear to be the infection’s main feature. Some reports have also included heart failure. ![]() Arrhythmias, which can be fatal, appear to be more common among hospitalized patients, although it’s unclear how frequently they’re related to myocarditis. But it can cause serious acute outcomes, including arrhythmias, heart failure, cardiac arrest, and sudden death.Īlthough a few case reports have emerged of fulminant myocarditis with cardiogenic shock-the most sudden and extreme form-during acute SARS-CoV-2 infection, the finding has been rare. Most often triggered by a virus, myocarditis usually resolves on its own as the infection wanes. But experts said that the most important question is the clinical one: What will COVID-19–associated heart injury mean, over the short-term and long-term, for the tens of millions of people around the world infected with the virus?Įarly in the pandemic, as reports documented elevated troponin levels in severe COVID-19 cases, suspicion quickly turned to myocarditis, an inflammatory disease in the heart muscle. Researchers are also working to explain the cardiac damage, with recent attention shifting from viral myocarditis to systemic inflammation. Physicians and scientists wondered then, as now: How common is heart injury across the spectrum of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection, from asymptomatic cases to critical disease? ![]() But so were those without a history of heart problems who had elevated troponin levels. It wasn’t surprising that patients with preexisting cardiovascular issues-prior heart failure, coronary artery disease, hypertension-were more likely to fare poorly, based on other respiratory illnesses. “It was quite clear that people that came into the hospital sick that had heart injury were the ones that were at greatest risk of requiring mechanical ventilation and, ultimately, at the greatest risk of dying,” said Aaron Baggish, MD, director of the cardiovascular performance program at Massachusetts General Hospital. ![]() Shared Decision Making and Communication.Scientific Discovery and the Future of Medicine.Health Care Economics, Insurance, Payment.Clinical Implications of Basic Neuroscience.Challenges in Clinical Electrocardiography. ![]()
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